Yae1p YJR067C

fungal protein found in Saccharomyces cerevisiae S288c
Protein protein Q27552827
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Yae1p YJR067C

Summary

Yae1p YJR067C is a protein[1].

Key Facts

  • Yae1p YJR067C's instance of is recorded as protein[2].
  • Yae1p YJR067C's subclass of is recorded as protein[3].
  • Yae1p YJR067C's UniProt protein ID is recorded as P47118[4].
  • Yae1p YJR067C's part of is recorded as Yae1-like[5].
  • Yae1p YJR067C's part of is recorded as Essential protein Yae1, N-terminal domain, protein family[6].
  • Yae1p YJR067C's has part is recorded as Essential protein Yae1, N-terminal[7].
  • Yae1p YJR067C's RefSeq protein ID is recorded as NP_012601[8].
  • Yae1p YJR067C's molecular function is recorded as protein binding[9].
  • Yae1p YJR067C's molecular function is recorded as molecular function[10].
  • Yae1p YJR067C's molecular function is recorded as protein-macromolecule adaptor activity[11].
  • Yae1p YJR067C's cell component is recorded as nucleus[12].
  • Yae1p YJR067C's cell component is recorded as cytoplasm[13].
  • Yae1p YJR067C's cell component is recorded as CIA complex[14].
  • Yae1p YJR067C's cell component is recorded as nucleus[15].
  • Yae1p YJR067C's cell component is recorded as cytoplasm[16].
  • Yae1p YJR067C's cell component is recorded as Yae1-Lto1 complex[17].
  • Yae1p YJR067C's biological process is recorded as translational initiation[18].
  • Yae1p YJR067C's biological process is recorded as ribosomal large subunit biogenesis[19].
  • Yae1p YJR067C's biological process is recorded as translational initiation[20].
  • Yae1p YJR067C's biological process is recorded as ribosomal large subunit biogenesis[21].
  • Yae1p YJR067C's biological process is recorded as protein maturation by [4Fe-4S] cluster transfer[22].
  • Yae1p YJR067C's biological process is recorded as translational initiation[23].
  • Yae1p YJR067C's biological process is recorded as ribosomal large subunit biogenesis[24].
  • Yae1p YJR067C's biological process is recorded as protein maturation by [4Fe-4S] cluster transfer[25].
  • Yae1p YJR067C's encoded by is recorded as YAE1[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q20641742. Retrieved . wikidata.org.
  3. [4] . Q905695. Retrieved . wikidata.org.
  4. [5] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] . wikidata.org.
  6. [7] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  7. [8] . Q20641742. Retrieved . wikidata.org.
  8. [9] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . The function of ORAOV1/LTO1, a gene that is overexpressed frequently in cancer: essential roles in the function and biogenesis of the ribosome. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . The function of ORAOV1/LTO1, a gene that is overexpressed frequently in cancer: essential roles in the function and biogenesis of the ribosome. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . The function of ORAOV1/LTO1, a gene that is overexpressed frequently in cancer: essential roles in the function and biogenesis of the ribosome. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . The function of ORAOV1/LTO1, a gene that is overexpressed frequently in cancer: essential roles in the function and biogenesis of the ribosome. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . The deca-GX3 proteins Yae1-Lto1 function as adaptors recruiting the ABC protein Rli1 for iron-sulfur cluster insertion.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . Q905695. Retrieved . wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Yae1p YJR067C. Retrieved May 3, 2026, from https://4ort.xyz/entity/yae1p-yjr067c
MLA “Yae1p YJR067C.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/yae1p-yjr067c.
BibTeX @misc{4ortxyz_yae1p-yjr067c_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Yae1p YJR067C}}, year = {2026}, url = {https://4ort.xyz/entity/yae1p-yjr067c}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Yae1p YJR067C — https://4ort.xyz/entity/yae1p-yjr067c (retrieved 2026-05-03)

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