X-ray repair cross complementing 5
mammalian protein found in Homo sapiens
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Summary
X-ray repair cross complementing 5 is a protein[1]. It is known by 19 alternative names across languages and contexts.[2]
Key Facts
- Part of: SPOC-like, C-terminal domain superfamily[3], Ku, C-terminal domain superfamily[4], von Willebrand factor A-like domain superfamily[5], Ku80[6], Ku, C-terminal domain, protein family[7], Ku70/Ku80, N-terminal alpha/beta, protein family[8], Ku70/Ku80 C-terminal arm, protein family[9], Ku70/Ku80 beta-barrel domain, protein family[10], von Willebrand factor, type A, protein family[11]
- Has part(s): Ku70/Ku80 C-terminal arm[12], Ku, C-terminal[13], von Willebrand factor, type A[14], Ku70/Ku80, N-terminal alpha/beta[15], Ku70/Ku80 beta-barrel domain[16]
- Molecular function: DNA binding[17], nucleotide binding[18], double-stranded telomeric DNA binding[19], telomeric DNA binding[20], helicase activity[21], 5'-deoxyribose-5-phosphate lyase activity[22], DNA helicase activity[23], hydrolase activity, acting on acid anhydrides[24], protein C-terminus binding[25], protein binding[26], hydrolase activity[27], ATP binding[28], ubiquitin protein ligase binding[29], double-stranded DNA binding[30]
- Cell component: cytoplasm[31], cytosol[32], nuclear telomere cap complex[33], membrane[34], plasma membrane[35], nucleoplasm[36], chromosome[37], nucleolus[38], nonhomologous end joining complex[39], nucleus[40], extracellular region[41], secretory granule lumen[42], protein-DNA complex[43], Ku70:Ku80 complex[44]
- Biological process: DNA recombination[45], regulation of transcription, DNA-templated[46], regulation of smooth muscle cell proliferation[47], transcription, DNA-templated[48], cellular response to DNA damage stimulus[49], positive regulation of neurogenesis[50], hematopoietic stem cell differentiation[51], double-strand break repair[52], negative regulation of t-circle formation[53], establishment of integrated proviral latency[54], cell population proliferation[55], negative regulation of transcription, DNA-templated[56], positive regulation of type I interferon production[57], cellular hyperosmotic salinity response[58]