Translation termination factor GTPase eRF3 YDR172W

fungal protein found in Saccharomyces cerevisiae S288c
Protein protein Q27552044
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Summary

Translation termination factor GTPase eRF3 YDR172W is a protein[1].

Key Facts

  • Translation termination factor GTPase eRF3 YDR172W's instance of is recorded as protein[2].
  • Translation termination factor GTPase eRF3 YDR172W's subclass of is recorded as protein[3].
  • Translation termination factor GTPase eRF3 YDR172W's UniProt protein ID is recorded as P05453[4].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as eukaryotic peptide chain release factor GTP-binding subunit[5].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Translation protein, beta-barrel domain superfamily[6].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Translation elongation factor EF1A/initiation factor IF2gamma, C-terminal[7].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as P-loop containing nucleoside triphosphate hydrolase[8].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Translation elongation factor EFTu/EF1A, C-terminal domain, protein family[9].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Translation elongation factor EFTu-like, domain 2, protein family[10].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Transcription factor, GTP-binding domain, protein family[11].
  • Translation termination factor GTPase eRF3 YDR172W's part of is recorded as Tr-type G domain, conserved site, protein family[12].
  • Translation termination factor GTPase eRF3 YDR172W's has part is recorded as translation elongation factor EFTu-like, domain 2[13].
  • Translation termination factor GTPase eRF3 YDR172W's has part is recorded as translation elongation factor EFTu/EF1A, C-terminal[14].
  • Translation termination factor GTPase eRF3 YDR172W's has part is recorded as Tr-type G domain, conserved site[15].
  • Translation termination factor GTPase eRF3 YDR172W's has part is recorded as transcription factor, GTP-binding domain[16].
  • Translation termination factor GTPase eRF3 YDR172W's RefSeq protein ID is recorded as NP_010457[17].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as GTPase activity[18].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as GTP binding[19].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as nucleotide binding[20].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as translation release factor activity[21].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as protein binding[22].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as identical protein binding[23].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as translation release factor activity[24].
  • Translation termination factor GTPase eRF3 YDR172W's molecular function is recorded as mRNA binding[25].
  • Translation termination factor GTPase eRF3 YDR172W's cell component is recorded as cytoplasmic stress granule[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] ↑ . UniProt. Retrieved . wikidata.org.
  2. [3] ↑ . UniProt. Retrieved . wikidata.org.
  3. [4] ↑ . UniProt. Retrieved . wikidata.org.
  4. [5] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  6. [7] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  7. [8] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  8. [9] ↑ . wikidata.org.
  9. [10] ↑ . wikidata.org.
  10. [11] ↑ . wikidata.org.
  11. [12] ↑ . wikidata.org.
  12. [13] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  13. [14] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  14. [15] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  15. [16] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  16. [17] ↑ . Q20641742. Retrieved . wikidata.org.
  17. [18] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] ↑ . Mtt1 is a Upf1-like helicase that interacts with the translation termination factors and whose overexpression can modulate termination efficiency. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] ↑ . Dominant prion mutants induce curing through pathways that promote chaperone-mediated disaggregation. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] ↑ . Global analysis of yeast mRNPs. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] ↑ . Heat Shock-Induced Accumulation of Translation Elongation and Termination Factors Precedes Assembly of Stress Granules in S. cerevisiae. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] ↑ . Wikidata. wikidata.org.

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APA 4ort.xyz Knowledge Graph. (2026). Translation termination factor GTPase eRF3 YDR172W. Retrieved October 22, 2026, from https://4ort.xyz/entity/translation-termination-factor-gtpase-erf3-ydr172w
MLA “Translation termination factor GTPase eRF3 YDR172W.” 4ort.xyz Knowledge Graph, 4ort.xyz, 22 Oct. 2026, https://4ort.xyz/entity/translation-termination-factor-gtpase-erf3-ydr172w.
BibTeX @misc{4ortxyz_translation-termination-factor-gtpase-erf3-ydr172w_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Translation termination factor GTPase eRF3 YDR172W}}, year = {2026}, url = {https://4ort.xyz/entity/translation-termination-factor-gtpase-erf3-ydr172w}, note = {Accessed: 2026-10-22}}
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