Transcription activation suppressor

mammalian protein found in Homo sapiens
Protein protein Q21121444
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Transcription activation suppressor

Summary

Transcription activation suppressor is a protein[1].

Key Facts

  • Transcription activation suppressor's instance of is recorded as protein[2].
  • Transcription activation suppressor's UniProt protein ID is recorded as Q9UK61[3].
  • Transcription activation suppressor's part of is recorded as Protein of unknown function DUF3715, protein family[4].
  • Transcription activation suppressor's has part is recorded as Protein of unknown function DUF3715[5].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001106207[6].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_056039[7].
  • Transcription activation suppressor's RefSeq protein ID is recorded as XP_006713141[8].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001350869[9].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001352564[10].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001352565[11].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001352566[12].
  • Transcription activation suppressor's RefSeq protein ID is recorded as NP_001352567[13].
  • Transcription activation suppressor's molecular function is recorded as RNA binding[14].
  • Transcription activation suppressor's molecular function is recorded as protein binding[15].
  • Transcription activation suppressor's molecular function is recorded as RNA binding[16].
  • Transcription activation suppressor's molecular function is recorded as chromatin binding[17].
  • Transcription activation suppressor's cell component is recorded as nucleus[18].
  • Transcription activation suppressor's cell component is recorded as chromosome[19].
  • Transcription activation suppressor's cell component is recorded as nucleoplasm[20].
  • Transcription activation suppressor's cell component is recorded as heterochromatin[21].
  • Transcription activation suppressor's biological process is recorded as regulation of transcription, DNA-templated[22].
  • Transcription activation suppressor's biological process is recorded as transcription, DNA-templated[23].
  • Transcription activation suppressor's biological process is recorded as negative regulation of gene expression, epigenetic[24].
  • Transcription activation suppressor's biological process is recorded as positive regulation of DNA methylation-dependent heterochromatin assembly[25].
  • Transcription activation suppressor's biological process is recorded as protein localization to heterochromatin[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] ↑ . Q905695. Retrieved . wikidata.org.
  2. [3] ↑ . Q905695. Retrieved . wikidata.org.
  3. [4] ↑ . wikidata.org.
  4. [5] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] ↑ . Q20641742. Retrieved . wikidata.org.
  6. [7] ↑ . Q20641742. Retrieved . wikidata.org.
  7. [8] ↑ . Q20641742. Retrieved . wikidata.org.
  8. [9] ↑ . Q20641742. Retrieved . wikidata.org.
  9. [10] ↑ . Q20641742. Retrieved . wikidata.org.
  10. [11] ↑ . Q20641742. Retrieved . wikidata.org.
  11. [12] ↑ . Q20641742. Retrieved . wikidata.org.
  12. [13] ↑ . Q20641742. Retrieved . wikidata.org.
  13. [14] ↑ . Insights into RNA biology from an atlas of mammalian mRNA-binding proteins. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] ↑ . Hyperactivation of HUSH complex function by Charcot-Marie-Tooth disease mutation in MORC2.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] ↑ . Insights into RNA biology from an atlas of mammalian mRNA-binding proteins. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] ↑ . Selective silencing of euchromatic L1s revealed by genome-wide screens for L1 regulators. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] ↑ . Hyperactivation of HUSH complex function by Charcot-Marie-Tooth disease mutation in MORC2.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] ↑ . GENE SILENCING. Epigenetic silencing by the HUSH complex mediates position-effect variegation in human cells.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] ↑ . Hyperactivation of HUSH complex function by Charcot-Marie-Tooth disease mutation in MORC2.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] ↑ . Hyperactivation of HUSH complex function by Charcot-Marie-Tooth disease mutation in MORC2.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] ↑ . Wikidata. wikidata.org.

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Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Transcription activation suppressor. Retrieved May 3, 2026, from https://4ort.xyz/entity/transcription-activation-suppressor
MLA “Transcription activation suppressor.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/transcription-activation-suppressor.
BibTeX @misc{4ortxyz_transcription-activation-suppressor_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Transcription activation suppressor}}, year = {2026}, url = {https://4ort.xyz/entity/transcription-activation-suppressor}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Transcription activation suppressor — https://4ort.xyz/entity/transcription-activation-suppressor (retrieved 2026-05-03)

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