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TCF3 fusion partner

mammalian protein found in Rattus norvegicus
Protein protein Q28558865
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TCF3 fusion partner

Summary

TCF3 fusion partner is a protein[1].

Key Facts

  • TCF3 fusion partner's instance of is recorded as protein[2].
  • TCF3 fusion partner's subclass of is recorded as protein[3].
  • TCF3 fusion partner's UniProt protein ID is recorded as Q9JMG6[4].
  • TCF3 fusion partner's part of is recorded as TCF3 fusion partner[5].
  • TCF3 fusion partner's RefSeq protein ID is recorded as NP_620225[6].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_006228098[7].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_006228099[8].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_038948867[9].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_038948868[10].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_038948869[11].
  • TCF3 fusion partner's RefSeq protein ID is recorded as XP_038948870[12].
  • TCF3 fusion partner's RefSeq protein ID is recorded as NP_001385582[13].
  • TCF3 fusion partner's RefSeq protein ID is recorded as NP_001385584[14].
  • TCF3 fusion partner's molecular function is recorded as DNA binding[15].
  • TCF3 fusion partner's molecular function is recorded as protein binding[16].
  • TCF3 fusion partner's molecular function is recorded as protein kinase binding[17].
  • TCF3 fusion partner's molecular function is recorded as protein heterodimerization activity[18].
  • TCF3 fusion partner's cell component is recorded as nucleus[19].
  • TCF3 fusion partner's cell component is recorded as cytoplasm[20].
  • TCF3 fusion partner's cell component is recorded as actin filament[21].
  • TCF3 fusion partner's cell component is recorded as Ino80 complex[22].
  • TCF3 fusion partner's biological process is recorded as DNA repair[23].
  • TCF3 fusion partner's biological process is recorded as DNA recombination[24].
  • TCF3 fusion partner's biological process is recorded as apoptotic process[25].
  • TCF3 fusion partner's biological process is recorded as cellular response to DNA damage stimulus[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] ↑ . Q905695. Retrieved . wikidata.org.
  2. [3] ↑ . Q905695. Retrieved . wikidata.org.
  3. [4] ↑ . Q905695. Retrieved . wikidata.org.
  4. [5] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] ↑ . Q20641742. Retrieved . wikidata.org.
  6. [7] ↑ . Q20641742. Retrieved . wikidata.org.
  7. [8] ↑ . Q20641742. Retrieved . wikidata.org.
  8. [9] ↑ . Q20641742. Retrieved . wikidata.org.
  9. [10] ↑ . Q20641742. Retrieved . wikidata.org.
  10. [11] ↑ . Q20641742. Retrieved . wikidata.org.
  11. [12] ↑ . Q20641742. Retrieved . wikidata.org.
  12. [13] ↑ . Q20641742. Retrieved . wikidata.org.
  13. [14] ↑ . Q20641742. Retrieved . wikidata.org.
  14. [15] ↑ . Arrest of cell cycle by amida which is phosphorylated by Cdc2 kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] ↑ . Par-4-mediated recruitment of Amida to the actin cytoskeleton leads to the induction of apoptosis. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] ↑ . Arrest of cell cycle by amida which is phosphorylated by Cdc2 kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] ↑ . Molecular cloning and characterization of Amida, a novel protein which interacts with a neuron-specific immediate early gene product arc, contains novel nuclear localization signals, and causes cell death in cultured cells. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] ↑ . Par-4-mediated recruitment of Amida to the actin cytoskeleton leads to the induction of apoptosis. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] ↑ . Par-4-mediated recruitment of Amida to the actin cytoskeleton leads to the induction of apoptosis. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] ↑ . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] ↑ . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). TCF3 fusion partner. Retrieved May 3, 2026, from https://4ort.xyz/entity/tcf3-fusion-partner-q28558865
MLA “TCF3 fusion partner.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/tcf3-fusion-partner-q28558865.
BibTeX @misc{4ortxyz_tcf3-fusion-partner-q28558865_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{TCF3 fusion partner}}, year = {2026}, url = {https://4ort.xyz/entity/tcf3-fusion-partner-q28558865}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): TCF3 fusion partner — https://4ort.xyz/entity/tcf3-fusion-partner-q28558865 (retrieved 2026-05-03)

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