Tailless Dmel_CG1378

protein found in Drosophila melanogaster
Protein protein Q29810425
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Tailless Dmel_CG1378

Summary

Tailless Dmel_CG1378 is a protein[1].

Key Facts

  • Tailless Dmel_CG1378's instance of is recorded as protein[2].
  • Tailless Dmel_CG1378's UniProt protein ID is recorded as P18102[3].
  • Tailless Dmel_CG1378's part of is recorded as Nuclear hormone receptor-like domain superfamily[4].
  • Tailless Dmel_CG1378's part of is recorded as Zinc finger, NHR/GATA-type[5].
  • Tailless Dmel_CG1378's part of is recorded as nuclear receptor[6].
  • Tailless Dmel_CG1378's part of is recorded as Nuclear hormone receptor, ligand-binding domain, protein family[7].
  • Tailless Dmel_CG1378's part of is recorded as Zinc finger, nuclear hormone receptor-type, protein family[8].
  • Tailless Dmel_CG1378's has part is recorded as Nuclear hormone receptor, ligand-binding domain[9].
  • Tailless Dmel_CG1378's has part is recorded as nuclear hormone receptor-type zinc finger[10].
  • Tailless Dmel_CG1378's RefSeq protein ID is recorded as NP_524596[11].
  • Tailless Dmel_CG1378's molecular function is recorded as DNA binding[12].
  • Tailless Dmel_CG1378's molecular function is recorded as DNA-binding transcription factor activity[13].
  • Tailless Dmel_CG1378's molecular function is recorded as transcription factor activity, RNA polymerase II distal enhancer sequence-specific binding[14].
  • Tailless Dmel_CG1378's molecular function is recorded as steroid hormone receptor activity[15].
  • Tailless Dmel_CG1378's molecular function is recorded as nuclear receptor activity[16].
  • Tailless Dmel_CG1378's molecular function is recorded as protein binding[17].
  • Tailless Dmel_CG1378's molecular function is recorded as zinc ion binding[18].
  • Tailless Dmel_CG1378's molecular function is recorded as sequence-specific DNA binding[19].
  • Tailless Dmel_CG1378's molecular function is recorded as metal ion binding[20].
  • Tailless Dmel_CG1378's molecular function is recorded as DNA-binding transcription factor activity, RNA polymerase II-specific[21].
  • Tailless Dmel_CG1378's cell component is recorded as nucleus[22].
  • Tailless Dmel_CG1378's biological process is recorded as cell fate specification[23].
  • Tailless Dmel_CG1378's biological process is recorded as Bolwig's organ morphogenesis[24].
  • Tailless Dmel_CG1378's biological process is recorded as insect visual primordium development[25].
  • Tailless Dmel_CG1378's biological process is recorded as inter-male aggressive behavior[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  6. [7] . wikidata.org.
  7. [8] . wikidata.org.
  8. [9] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . Q20641742. Retrieved . wikidata.org.
  11. [12] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . Posterior stripe expression of hunchback is driven from two promoters by a common enhancer element.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . Comparison of complete nuclear receptor sets from the human, Caenorhabditis elegans and Drosophila genomes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . Drosophila brakeless interacts with atrophin and is required for tailless-mediated transcriptional repression in early embryos. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . Posterior stripe expression of hunchback is driven from two promoters by a common enhancer element.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . Developmental genetics of the Drosophila gut: specification of primordia, subdivision and overt-differentiation.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . Transcriptional regulation of atonal required for Drosophila larval eye development by concerted action of eyes absent, sine oculis and hedgehog signaling independent of fused kinase and cubitus interruptus.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . The control of cell fate in the embryonic visual system by atonal, tailless and EGFR signaling.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . Tailless and Atrophin control Drosophila aggression by regulating neuropeptide signalling in the pars intercerebralis. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

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Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Tailless Dmel_CG1378. Retrieved May 3, 2026, from https://4ort.xyz/entity/tailless-dmel-cg1378
MLA “Tailless Dmel_CG1378.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/tailless-dmel-cg1378.
BibTeX @misc{4ortxyz_tailless-dmel-cg1378_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Tailless Dmel_CG1378}}, year = {2026}, url = {https://4ort.xyz/entity/tailless-dmel-cg1378}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Tailless Dmel_CG1378 — https://4ort.xyz/entity/tailless-dmel-cg1378 (retrieved 2026-05-03)

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