Rumi Dmel_CG31152

protein found in Drosophila melanogaster
Protein protein Q29820884
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Rumi Dmel_CG31152

Summary

Rumi Dmel_CG31152 is a protein[1].

Key Facts

  • Rumi Dmel_CG31152's instance of is recorded as protein[2].
  • Rumi Dmel_CG31152's UniProt protein ID is recorded as Q8T045[3].
  • Rumi Dmel_CG31152's part of is recorded as Lipopolysaccharide-modifying protein[4].
  • Rumi Dmel_CG31152's RefSeq protein ID is recorded as NP_001262849[5].
  • Rumi Dmel_CG31152's RefSeq protein ID is recorded as NP_651095[6].
  • Rumi Dmel_CG31152's molecular function is recorded as transferase activity[7].
  • Rumi Dmel_CG31152's molecular function is recorded as glycosyltransferase activity[8].
  • Rumi Dmel_CG31152's molecular function is recorded as UDP-glucosyltransferase activity[9].
  • Rumi Dmel_CG31152's molecular function is recorded as UDP-xylosyltransferase activity[10].
  • Rumi Dmel_CG31152's molecular function is recorded as glucosyltransferase activity[11].
  • Rumi Dmel_CG31152's molecular function is recorded as UDP-glucosyltransferase activity[12].
  • Rumi Dmel_CG31152's molecular function is recorded as glucosyltransferase activity[13].
  • Rumi Dmel_CG31152's cell component is recorded as endoplasmic reticulum[14].
  • Rumi Dmel_CG31152's cell component is recorded as endoplasmic reticulum lumen[15].
  • Rumi Dmel_CG31152's cell component is recorded as endomembrane system[16].
  • Rumi Dmel_CG31152's cell component is recorded as endoplasmic reticulum lumen[17].
  • Rumi Dmel_CG31152's cell component is recorded as endomembrane system[18].
  • Rumi Dmel_CG31152's biological process is recorded as protein glycosylation[19].
  • Rumi Dmel_CG31152's biological process is recorded as protein O-linked glycosylation[20].
  • Rumi Dmel_CG31152's biological process is recorded as glycolipid metabolic process[21].
  • Rumi Dmel_CG31152's biological process is recorded as Golgi organization[22].
  • Rumi Dmel_CG31152's biological process is recorded as Notch signaling pathway[23].
  • Rumi Dmel_CG31152's biological process is recorded as rhabdomere development[24].
  • Rumi Dmel_CG31152's biological process is recorded as negative regulation of Notch signaling pathway[25].
  • Rumi Dmel_CG31152's biological process is recorded as positive regulation of Notch signaling pathway[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] . Q20641742. Retrieved . wikidata.org.
  5. [6] . Q20641742. Retrieved . wikidata.org.
  6. [7] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  7. [8] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  8. [9] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . Rumi is a CAP10 domain glycosyltransferase that modifies Notch and is required for Notch signaling.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . Structural analysis of Notch-regulating Rumi reveals basis for pathogenic mutations. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . Rumi is a CAP10 domain glycosyltransferase that modifies Notch and is required for Notch signaling.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . Mapping organelle proteins and protein complexes in Drosophila melanogaster. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . Mapping organelle proteins and protein complexes in Drosophila melanogaster. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . Identification of ER proteins involved in the functional organisation of the early secretory pathway in Drosophila cells by a targeted RNAi screen. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . Rumi is a CAP10 domain glycosyltransferase that modifies Notch and is required for Notch signaling.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . The protein O-glucosyltransferase Rumi modifies eyes shut to promote rhabdomere separation in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . Rumi is a CAP10 domain glycosyltransferase that modifies Notch and is required for Notch signaling.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . A POGLUT1 mutation causes a muscular dystrophy with reduced Notch signaling and satellite cell loss.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Rumi Dmel_CG31152. Retrieved May 3, 2026, from https://4ort.xyz/entity/rumi-dmel-cg31152
MLA “Rumi Dmel_CG31152.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/rumi-dmel-cg31152.
BibTeX @misc{4ortxyz_rumi-dmel-cg31152_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Rumi Dmel_CG31152}}, year = {2026}, url = {https://4ort.xyz/entity/rumi-dmel-cg31152}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Rumi Dmel_CG31152 — https://4ort.xyz/entity/rumi-dmel-cg31152 (retrieved 2026-05-03)

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