Npr3p YHL023C
fungal protein found in Saccharomyces cerevisiae S288c
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Npr3p YHL023C
Summary
Npr3p YHL023C is a protein[1].
Key Facts
- Npr3p YHL023C's instance of is recorded as protein[2].
- Npr3p YHL023C's UniProt protein ID is recorded as P38742[3].
- Npr3p YHL023C's part of is recorded as Nitrogen permease regulator 3[4].
- Npr3p YHL023C's part of is recorded as membrane protein[5].
- Npr3p YHL023C's RefSeq protein ID is recorded as NP_011840[6].
- Npr3p YHL023C's molecular function is recorded as protein binding[7].
- Npr3p YHL023C's molecular function is recorded as molecular function[8].
- Npr3p YHL023C's cell component is recorded as membrane[9].
- Npr3p YHL023C's cell component is recorded as vacuolar membrane[10].
- Npr3p YHL023C's cell component is recorded as GATOR1 complex[11].
- Npr3p YHL023C's cell component is recorded as extrinsic component of fungal-type vacuolar membrane[12].
- Npr3p YHL023C's cell component is recorded as vacuole[13].
- Npr3p YHL023C's cell component is recorded as Seh1-associated complex[14].
- Npr3p YHL023C's cell component is recorded as extrinsic component of fungal-type vacuolar membrane[15].
- Npr3p YHL023C's cell component is recorded as GATOR1 complex[16].
- Npr3p YHL023C's biological process is recorded as negative regulation of TOR signaling[17].
- Npr3p YHL023C's biological process is recorded as negative regulation of small GTPase mediated signal transduction[18].
- Npr3p YHL023C's biological process is recorded as positive regulation of autophagy[19].
- Npr3p YHL023C's biological process is recorded as regulation of autophagosome assembly[20].
- Npr3p YHL023C's biological process is recorded as positive regulation of cellular response to amino acid starvation[21].
- Npr3p YHL023C's biological process is recorded as pseudohyphal growth[22].
- Npr3p YHL023C's biological process is recorded as protein transport[23].
- Npr3p YHL023C's biological process is recorded as TORC1 signaling[24].
- Npr3p YHL023C's biological process is recorded as meiosis[25].
- Npr3p YHL023C's biological process is recorded as negative regulation of TOR signaling[26].