ETS-domain lacking Dmel_CG15085

protein found in Drosophila melanogaster
Protein protein Q29817032
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ETS-domain lacking Dmel_CG15085

Summary

ETS-domain lacking Dmel_CG15085 is a protein[1].

Key Facts

  • ETS-domain lacking Dmel_CG15085's instance of is recorded as protein[2].
  • ETS-domain lacking Dmel_CG15085's UniProt protein ID is recorded as Q7K119[3].
  • ETS-domain lacking Dmel_CG15085's part of is recorded as Sterile alpha motif/pointed domain superfamily[4].
  • ETS-domain lacking Dmel_CG15085's part of is recorded as Pointed domain, protein family[5].
  • ETS-domain lacking Dmel_CG15085's has part is recorded as Pointed domain[6].
  • ETS-domain lacking Dmel_CG15085's RefSeq protein ID is recorded as NP_523786[7].
  • ETS-domain lacking Dmel_CG15085's molecular function is recorded as DNA-binding transcription factor activity, RNA polymerase II-specific[8].
  • ETS-domain lacking Dmel_CG15085's molecular function is recorded as protein binding[9].
  • ETS-domain lacking Dmel_CG15085's molecular function is recorded as sequence-specific DNA binding[10].
  • ETS-domain lacking Dmel_CG15085's cell component is recorded as nucleus[11].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as negative regulation of transcription by RNA polymerase II[12].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of transcription, DNA-templated[13].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of transcription by RNA polymerase II[14].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as sensory organ development[15].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as embryonic pattern specification[16].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of epidermal growth factor receptor signaling pathway[17].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of phosphorylation[18].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as photoreceptor cell development[19].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as negative regulation of transcription, DNA-templated[20].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of Ras protein signal transduction[21].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as nuclear export[22].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as negative regulation of epidermal growth factor receptor signaling pathway[23].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as negative regulation of DNA-binding transcription factor activity[24].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of DNA-binding transcription factor activity[25].
  • ETS-domain lacking Dmel_CG15085's biological process is recorded as regulation of transcription by RNA polymerase II[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] . wikidata.org.
  5. [6] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  6. [7] . Q20641742. Retrieved . wikidata.org.
  7. [8] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  8. [9] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . EDL/MAE regulates EGF-mediated induction by antagonizing Ets transcription factor Pointed. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . EDL/MAE regulates EGF-mediated induction by antagonizing Ets transcription factor Pointed. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . EDL/MAE regulates EGF-mediated induction by antagonizing Ets transcription factor Pointed. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . Antagonistic regulation of Yan nuclear export by Mae and Crm1 may increase the stringency of the Ras response. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . EDL/MAE regulates EGF-mediated induction by antagonizing Ets transcription factor Pointed. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . EDL/MAE regulates EGF-mediated induction by antagonizing Ets transcription factor Pointed. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . Mae mediates MAP kinase phosphorylation of Ets transcription factors in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). ETS-domain lacking Dmel_CG15085. Retrieved May 3, 2026, from https://4ort.xyz/entity/ets-domain-lacking-dmel-cg15085
MLA “ETS-domain lacking Dmel_CG15085.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/ets-domain-lacking-dmel-cg15085.
BibTeX @misc{4ortxyz_ets-domain-lacking-dmel-cg15085_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{ETS-domain lacking Dmel_CG15085}}, year = {2026}, url = {https://4ort.xyz/entity/ets-domain-lacking-dmel-cg15085}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): ETS-domain lacking Dmel_CG15085 — https://4ort.xyz/entity/ets-domain-lacking-dmel-cg15085 (retrieved 2026-05-03)

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