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Disabled Dmel_CG9695

protein found in Drosophila melanogaster
Protein protein Q29814627
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Disabled Dmel_CG9695

Summary

Disabled Dmel_CG9695 is a protein[1].

Key Facts

  • Disabled Dmel_CG9695's instance of is recorded as protein[2].
  • Disabled Dmel_CG9695's UniProt protein ID is recorded as P98081[3].
  • Disabled Dmel_CG9695's part of is recorded as Protein disabled[4].
  • Disabled Dmel_CG9695's part of is recorded as PH-like domain superfamily[5].
  • Disabled Dmel_CG9695's part of is recorded as PTB/PI domain, protein family[6].
  • Disabled Dmel_CG9695's has part is recorded as PTB/PI domain[7].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001163455[8].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001246800[9].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001246801[10].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001246802[11].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001261967[12].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001261968[13].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_001261969[14].
  • Disabled Dmel_CG9695's RefSeq protein ID is recorded as NP_524119[15].
  • Disabled Dmel_CG9695's molecular function is recorded as sevenless binding[16].
  • Disabled Dmel_CG9695's molecular function is recorded as protein binding[17].
  • Disabled Dmel_CG9695's molecular function is recorded as SH3 domain binding[18].
  • Disabled Dmel_CG9695's molecular function is recorded as SH2 domain binding[19].
  • Disabled Dmel_CG9695's molecular function is recorded as SH3 domain binding[20].
  • Disabled Dmel_CG9695's cell component is recorded as cytoplasm[21].
  • Disabled Dmel_CG9695's cell component is recorded as axon[22].
  • Disabled Dmel_CG9695's cell component is recorded as synapse[23].
  • Disabled Dmel_CG9695's cell component is recorded as cytoplasm[24].
  • Disabled Dmel_CG9695's cell component is recorded as cytosol[25].
  • Disabled Dmel_CG9695's cell component is recorded as presynapse[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] ↑ . Q905695. Retrieved . wikidata.org.
  2. [3] ↑ . Q905695. Retrieved . wikidata.org.
  3. [4] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] ↑ . wikidata.org.
  6. [7] ↑ . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  7. [8] ↑ . Q20641742. Retrieved . wikidata.org.
  8. [9] ↑ . Q20641742. Retrieved . wikidata.org.
  9. [10] ↑ . Q20641742. Retrieved . wikidata.org.
  10. [11] ↑ . Q20641742. Retrieved . wikidata.org.
  11. [12] ↑ . Q20641742. Retrieved . wikidata.org.
  12. [13] ↑ . Q20641742. Retrieved . wikidata.org.
  13. [14] ↑ . Q20641742. Retrieved . wikidata.org.
  14. [15] ↑ . Q20641742. Retrieved . wikidata.org.
  15. [16] ↑ . Disabled Is a Putative Adaptor Protein That Functions during Signaling by the Sevenless Receptor Tyrosine Kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] ↑ . Disabled Is a Putative Adaptor Protein That Functions during Signaling by the Sevenless Receptor Tyrosine Kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] ↑ . Disabled Is a Putative Adaptor Protein That Functions during Signaling by the Sevenless Receptor Tyrosine Kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] ↑ . Disabled Is a Putative Adaptor Protein That Functions during Signaling by the Sevenless Receptor Tyrosine Kinase. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] ↑ . Dosage-sensitive modifiers of Drosophila abl tyrosine kinase function: prospero, a regulator of axonal outgrowth, and disabled, a novel tyrosine kinase substrate.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] ↑ . Dosage-sensitive modifiers of Drosophila abl tyrosine kinase function: prospero, a regulator of axonal outgrowth, and disabled, a novel tyrosine kinase substrate.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] ↑ . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Disabled Dmel_CG9695. Retrieved May 3, 2026, from https://4ort.xyz/entity/disabled-dmel-cg9695
MLA “Disabled Dmel_CG9695.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/disabled-dmel-cg9695.
BibTeX @misc{4ortxyz_disabled-dmel-cg9695_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Disabled Dmel_CG9695}}, year = {2026}, url = {https://4ort.xyz/entity/disabled-dmel-cg9695}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Disabled Dmel_CG9695 — https://4ort.xyz/entity/disabled-dmel-cg9695 (retrieved 2026-05-03)

Canonical URL: https://4ort.xyz/entity/disabled-dmel-cg9695 · Last refreshed: