Deterin Dmel_CG12265

protein found in Drosophila melanogaster
Protein protein Q29809854
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Deterin Dmel_CG12265

Summary

Deterin Dmel_CG12265 is a protein[1].

Key Facts

  • Deterin Dmel_CG12265's instance of is recorded as protein[2].
  • Deterin Dmel_CG12265's UniProt protein ID is recorded as Q9VEM2[3].
  • Deterin Dmel_CG12265's part of is recorded as BIR repeat domain, protein family[4].
  • Deterin Dmel_CG12265's has part is recorded as BIR repeat[5].
  • Deterin Dmel_CG12265's RefSeq protein ID is recorded as NP_650608[6].
  • Deterin Dmel_CG12265's molecular function is recorded as ubiquitin-protein transferase activity[7].
  • Deterin Dmel_CG12265's molecular function is recorded as cysteine-type endopeptidase inhibitor activity[8].
  • Deterin Dmel_CG12265's molecular function is recorded as zinc ion binding[9].
  • Deterin Dmel_CG12265's cell component is recorded as chromosome, centromeric region[10].
  • Deterin Dmel_CG12265's cell component is recorded as kinetochore[11].
  • Deterin Dmel_CG12265's cell component is recorded as nucleus[12].
  • Deterin Dmel_CG12265's cell component is recorded as cytoplasm[13].
  • Deterin Dmel_CG12265's cell component is recorded as chromosome passenger complex[14].
  • Deterin Dmel_CG12265's cell component is recorded as mitotic spindle midzone[15].
  • Deterin Dmel_CG12265's cell component is recorded as meiotic spindle midzone[16].
  • Deterin Dmel_CG12265's biological process is recorded as mitotic cytokinesis[17].
  • Deterin Dmel_CG12265's biological process is recorded as actomyosin contractile ring assembly[18].
  • Deterin Dmel_CG12265's biological process is recorded as mitotic spindle organization[19].
  • Deterin Dmel_CG12265's biological process is recorded as meiosis I cytokinesis[20].
  • Deterin Dmel_CG12265's biological process is recorded as meiosis II cytokinesis[21].
  • Deterin Dmel_CG12265's biological process is recorded as regulation of signal transduction[22].
  • Deterin Dmel_CG12265's biological process is recorded as protein ubiquitination[23].
  • Deterin Dmel_CG12265's biological process is recorded as meiotic cytokinesis[24].
  • Deterin Dmel_CG12265's biological process is recorded as negative regulation of apoptotic process[25].
  • Deterin Dmel_CG12265's biological process is recorded as meiotic chromosome segregation[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . wikidata.org.
  4. [5] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] . Q20641742. Retrieved . wikidata.org.
  6. [7] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  7. [8] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  8. [9] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . Genes involved in centrosome-independent mitotic spindle assembly in Drosophila S2 cells. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . Deterin, a new inhibitor of apoptosis from Drosophila melanogaster. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . The chromosomal passenger complex controls the function of endosomal sorting complex required for transport-III Snf7 proteins during cytokinesis. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . Genetic dissection of meiotic cytokinesis in Drosophila males.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . Genes involved in centrosome-independent mitotic spindle assembly in Drosophila S2 cells. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . Genetic dissection of meiotic cytokinesis in Drosophila males.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . Genetic dissection of meiotic cytokinesis in Drosophila males.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . A comparison of programmed cell death between species. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . Role of Survivin in cytokinesis revealed by a separation-of-function allele. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Deterin Dmel_CG12265. Retrieved May 3, 2026, from https://4ort.xyz/entity/deterin-dmel-cg12265
MLA “Deterin Dmel_CG12265.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/deterin-dmel-cg12265.
BibTeX @misc{4ortxyz_deterin-dmel-cg12265_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Deterin Dmel_CG12265}}, year = {2026}, url = {https://4ort.xyz/entity/deterin-dmel-cg12265}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Deterin Dmel_CG12265 — https://4ort.xyz/entity/deterin-dmel-cg12265 (retrieved 2026-05-03)

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