Cdc37 Dmel_CG12019

protein found in Drosophila melanogaster
Protein protein Q29815837
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Cdc37 Dmel_CG12019

Summary

Cdc37 Dmel_CG12019 is a protein[1].

Key Facts

  • Cdc37 Dmel_CG12019's instance of is recorded as protein[2].
  • Cdc37 Dmel_CG12019's UniProt protein ID is recorded as La Massana[3].
  • Cdc37 Dmel_CG12019's part of is recorded as Cdc37[4].
  • Cdc37 Dmel_CG12019's part of is recorded as Cdc37, Hsp90-binding domain superfamily[5].
  • Cdc37 Dmel_CG12019's part of is recorded as Cdc37, N-terminal domain, protein family[6].
  • Cdc37 Dmel_CG12019's part of is recorded as Cdc37, C-terminal domain, protein family[7].
  • Cdc37 Dmel_CG12019's part of is recorded as Cdc37, Hsp90 binding domain, protein family[8].
  • Cdc37 Dmel_CG12019's has part is recorded as Cdc37, C-terminal[9].
  • Cdc37 Dmel_CG12019's has part is recorded as Cdc37, Hsp90 binding[10].
  • Cdc37 Dmel_CG12019's has part is recorded as Cdc37, N-terminal domain[11].
  • Cdc37 Dmel_CG12019's RefSeq protein ID is recorded as NP_477006[12].
  • Cdc37 Dmel_CG12019's molecular function is recorded as protein binding[13].
  • Cdc37 Dmel_CG12019's molecular function is recorded as protein kinase binding[14].
  • Cdc37 Dmel_CG12019's molecular function is recorded as protein tyrosine kinase activator activity[15].
  • Cdc37 Dmel_CG12019's molecular function is recorded as heat shock protein binding[16].
  • Cdc37 Dmel_CG12019's molecular function is recorded as unfolded protein binding[17].
  • Cdc37 Dmel_CG12019's molecular function is recorded as chaperone binding[18].
  • Cdc37 Dmel_CG12019's cell component is recorded as cytoplasm[19].
  • Cdc37 Dmel_CG12019's biological process is recorded as protein folding[20].
  • Cdc37 Dmel_CG12019's biological process is recorded as cell cycle[21].
  • Cdc37 Dmel_CG12019's biological process is recorded as chromosome segregation[22].
  • Cdc37 Dmel_CG12019's biological process is recorded as multicellular organism development[23].
  • Cdc37 Dmel_CG12019's biological process is recorded as intracellular signal transduction[24].
  • Cdc37 Dmel_CG12019's biological process is recorded as positive regulation of Ras protein signal transduction[25].
  • Cdc37 Dmel_CG12019's biological process is recorded as protein stabilization[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  5. [6] . wikidata.org.
  6. [7] . wikidata.org.
  7. [8] . wikidata.org.
  8. [9] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  9. [10] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . Q20641742. Retrieved . wikidata.org.
  12. [13] . Modularity and hormone sensitivity of the Drosophila melanogaster insulin receptor/target of rapamycin interaction proteome. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . Mutations in Hsp83 and cdc37 impair signaling by the sevenless receptor tyrosine kinase in Drosophila.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . Mutations in Hsp83 and cdc37 impair signaling by the sevenless receptor tyrosine kinase in Drosophila.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . Mutations in Hsp83 and cdc37 impair signaling by the sevenless receptor tyrosine kinase in Drosophila.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . Mutations in Hsp83 and cdc37 impair signaling by the sevenless receptor tyrosine kinase in Drosophila.. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . A functional screen reveals an extensive layer of transcriptional and splicing control underlying RAS/MAPK signaling in Drosophila. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Cdc37 Dmel_CG12019. Retrieved May 3, 2026, from https://4ort.xyz/entity/cdc37-dmel-cg12019
MLA “Cdc37 Dmel_CG12019.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/cdc37-dmel-cg12019.
BibTeX @misc{4ortxyz_cdc37-dmel-cg12019_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Cdc37 Dmel_CG12019}}, year = {2026}, url = {https://4ort.xyz/entity/cdc37-dmel-cg12019}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Cdc37 Dmel_CG12019 — https://4ort.xyz/entity/cdc37-dmel-cg12019 (retrieved 2026-05-03)

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