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CAMP responsive element binding protein-like 2

mammalian protein found in Mus musculus
Protein protein Q21499357
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CAMP responsive element binding protein-like 2

Summary

CAMP responsive element binding protein-like 2 is a protein[1].

Key Facts

  • CAMP responsive element binding protein-like 2's instance of is recorded as protein[2].
  • CAMP responsive element binding protein-like 2's subclass of is recorded as protein[3].
  • CAMP responsive element binding protein-like 2's UniProt protein ID is recorded as Q32M00[4].
  • CAMP responsive element binding protein-like 2's RefSeq protein ID is recorded as NP_808355[5].
  • CAMP responsive element binding protein-like 2's RefSeq protein ID is recorded as XP_011239631[6].
  • CAMP responsive element binding protein-like 2's RefSeq protein ID is recorded as XP_036021980[7].
  • CAMP responsive element binding protein-like 2's molecular function is recorded as DNA-binding transcription factor activity[8].
  • CAMP responsive element binding protein-like 2's molecular function is recorded as DNA binding[9].
  • CAMP responsive element binding protein-like 2's molecular function is recorded as protein binding[10].
  • CAMP responsive element binding protein-like 2's cell component is recorded as nucleus[11].
  • CAMP responsive element binding protein-like 2's biological process is recorded as positive regulation of transcription, DNA-templated[12].
  • CAMP responsive element binding protein-like 2's biological process is recorded as positive regulation of glucose import[13].
  • CAMP responsive element binding protein-like 2's biological process is recorded as cell differentiation[14].
  • CAMP responsive element binding protein-like 2's biological process is recorded as protein stabilization[15].
  • CAMP responsive element binding protein-like 2's biological process is recorded as positive regulation of lipid biosynthetic process[16].
  • CAMP responsive element binding protein-like 2's biological process is recorded as regulation of transcription, DNA-templated[17].
  • CAMP responsive element binding protein-like 2's biological process is recorded as positive regulation of fat cell differentiation[18].
  • CAMP responsive element binding protein-like 2's biological process is recorded as transcription, DNA-templated[19].
  • CAMP responsive element binding protein-like 2's biological process is recorded as positive regulation of peptidyl-serine phosphorylation[20].
  • CAMP responsive element binding protein-like 2's encoded by is recorded as Crebl2[21].
  • CAMP responsive element binding protein-like 2's found in taxon is recorded as house mouse[22].
  • CAMP responsive element binding protein-like 2's Ensembl protein ID is recorded as ENSMUSP00000035304[23].
  • CAMP responsive element binding protein-like 2's Ensembl protein ID is recorded as ENSMUSP00000107568[24].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] ↑ . Q905695. Retrieved . wikidata.org.
  2. [3] ↑ . Q905695. Retrieved . wikidata.org.
  3. [4] ↑ . Q905695. Retrieved . wikidata.org.
  4. [5] ↑ . Q20641742. Retrieved . wikidata.org.
  5. [6] ↑ . Q20641742. Retrieved . wikidata.org.
  6. [7] ↑ . Q20641742. Retrieved . wikidata.org.
  7. [8] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  8. [9] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  9. [10] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] ↑ . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] ↑ . CREBL2, interacting with CREB, induces adipogenesis in 3T3-L1 adipocytes. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] ↑ . Q905695. Retrieved . wikidata.org.
  21. [22] ↑ . Q905695. Retrieved . wikidata.org.
  22. [23] ↑ . ensembl Release 106. wikidata.org.
  23. [24] ↑ . ensembl Release 106. wikidata.org.

Class ancestry

  1. [1] ↑ . Wikidata. wikidata.org.

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Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). CAMP responsive element binding protein-like 2. Retrieved May 3, 2026, from https://4ort.xyz/entity/camp-responsive-element-binding-protein-like-2-q21499357
MLA “CAMP responsive element binding protein-like 2.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/camp-responsive-element-binding-protein-like-2-q21499357.
BibTeX @misc{4ortxyz_camp-responsive-element-binding-protein-like-2-q21499357_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{CAMP responsive element binding protein-like 2}}, year = {2026}, url = {https://4ort.xyz/entity/camp-responsive-element-binding-protein-like-2-q21499357}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): CAMP responsive element binding protein-like 2 — https://4ort.xyz/entity/camp-responsive-element-binding-protein-like-2-q21499357 (retrieved 2026-05-03)

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