Actin alpha cardiac muscle 1

mammalian protein found in Homo sapiens
Protein protein Q21096423
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Actin alpha cardiac muscle 1

Summary

Actin alpha cardiac muscle 1 is a protein[1].

Key Facts

  • Actin alpha cardiac muscle 1's instance of is recorded as protein[2].
  • Actin alpha cardiac muscle 1's UniProt protein ID is recorded as P68032[3].
  • Actin alpha cardiac muscle 1's part of is recorded as actin[4].
  • Actin alpha cardiac muscle 1's part of is recorded as Actin/actin-like conserved site, protein family[5].
  • Actin alpha cardiac muscle 1's part of is recorded as Actin, conserved site, protein family[6].
  • Actin alpha cardiac muscle 1's has part is recorded as Actin, conserved site[7].
  • Actin alpha cardiac muscle 1's has part is recorded as Actin/actin-like conserved site[8].
  • Actin alpha cardiac muscle 1's RefSeq protein ID is recorded as NP_005150[9].
  • Actin alpha cardiac muscle 1's molecular function is recorded as nucleotide binding[10].
  • Actin alpha cardiac muscle 1's molecular function is recorded as myosin binding[11].
  • Actin alpha cardiac muscle 1's molecular function is recorded as ATPase activity[12].
  • Actin alpha cardiac muscle 1's molecular function is recorded as ATP binding[13].
  • Actin alpha cardiac muscle 1's molecular function is recorded as myosin binding[14].
  • Actin alpha cardiac muscle 1's cell component is recorded as cytoplasm[15].
  • Actin alpha cardiac muscle 1's cell component is recorded as cell body[16].
  • Actin alpha cardiac muscle 1's cell component is recorded as cytosol[17].
  • Actin alpha cardiac muscle 1's cell component is recorded as blood microparticle[18].
  • Actin alpha cardiac muscle 1's cell component is recorded as membrane[19].
  • Actin alpha cardiac muscle 1's cell component is recorded as I band[20].
  • Actin alpha cardiac muscle 1's cell component is recorded as focal adhesion[21].
  • Actin alpha cardiac muscle 1's cell component is recorded as filopodium[22].
  • Actin alpha cardiac muscle 1's cell component is recorded as sarcomere[23].
  • Actin alpha cardiac muscle 1's cell component is recorded as actin filament[24].
  • Actin alpha cardiac muscle 1's cell component is recorded as extracellular exosome[25].
  • Actin alpha cardiac muscle 1's cell component is recorded as cytoskeleton[26].

References

Programmatic citations — every numbered marker resolves to a verifiable graph row below.

Direct Wikidata claims

  1. [2] . Q905695. Retrieved . wikidata.org.
  2. [3] . Q905695. Retrieved . wikidata.org.
  3. [4] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  4. [5] . wikidata.org.
  5. [6] . wikidata.org.
  6. [7] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  7. [8] . InterPro Release 71.0. ebi.ac.uk. Provenance: wikidata.org.
  8. [9] . Q20641742. Retrieved . wikidata.org.
  9. [10] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  10. [11] . Functional consequences of a mutation in an expressed human alpha-cardiac actin at a site implicated in familial hypertrophic cardiomyopathy. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  11. [12] . Functional consequences of a mutation in an expressed human alpha-cardiac actin at a site implicated in familial hypertrophic cardiomyopathy. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  12. [13] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  13. [14] . Functional consequences of a mutation in an expressed human alpha-cardiac actin at a site implicated in familial hypertrophic cardiomyopathy. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  14. [15] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  15. [16] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  16. [17] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  17. [18] . Proteomic analysis of microvesicles from plasma of healthy donors reveals high individual variability. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  18. [19] . Defining the membrane proteome of NK cells. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  19. [20] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  20. [21] . Analysis of the myosin-II-responsive focal adhesion proteome reveals a role for β-Pix in negative regulation of focal adhesion maturation. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  21. [22] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  22. [23] . Defining alpha-skeletal and alpha-cardiac actin expression in human heart and skeletal muscle explains the absence of cardiac involvement in ACTA1 nemaline myopathy. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  23. [24] . Functional consequences of a mutation in an expressed human alpha-cardiac actin at a site implicated in familial hypertrophic cardiomyopathy. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  24. [25] . In-depth proteomic analyses of exosomes isolated from expressed prostatic secretions in urine. Retrieved . ebi.ac.uk. Provenance: wikidata.org.
  25. [26] . GOA. Retrieved . ebi.ac.uk. Provenance: wikidata.org.

Class ancestry

  1. [1] . Wikidata. wikidata.org.

📑 Cite this page

Use these citations when quoting this entity in research, articles, AI prompts, or wherever provenance matters. We aggregate Wikidata + Wikipedia + authoritative open-data sources; the stitched, scored, cross-referenced view is what 4ort.xyz contributes.

APA 4ort.xyz Knowledge Graph. (2026). Actin alpha cardiac muscle 1. Retrieved May 3, 2026, from https://4ort.xyz/entity/actin-alpha-cardiac-muscle-1
MLA “Actin alpha cardiac muscle 1.” 4ort.xyz Knowledge Graph, 4ort.xyz, 3 May. 2026, https://4ort.xyz/entity/actin-alpha-cardiac-muscle-1.
BibTeX @misc{4ortxyz_actin-alpha-cardiac-muscle-1_2026, author = {{4ort.xyz Knowledge Graph}}, title = {{Actin alpha cardiac muscle 1}}, year = {2026}, url = {https://4ort.xyz/entity/actin-alpha-cardiac-muscle-1}, note = {Accessed: 2026-05-03}}
LLM prompt According to 4ort.xyz Knowledge Graph (aggregator of Wikidata, Wikipedia, and authoritative open-data sources): Actin alpha cardiac muscle 1 — https://4ort.xyz/entity/actin-alpha-cardiac-muscle-1 (retrieved 2026-05-03)

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