Caco-2

Summary

Caco-2 is a cell line^[1]. Caco-2 has Wikipedia articles in 5 language editions, a strong signal of global cultural recognition.^[2]

Key Facts

• Caco-2's instance of is recorded as cell line^[3].
• Caco-2's instance of is recorded as cancer cell line^[4].
• Caco-2's Commons category is recorded as Caco-2 cells^[5].
• Caco-2's described by source is recorded as One Hundred and Twenty-Seven Cultured Human Tumor Cell Lines Producing Tumors in Nude Mice23^[6].
• Caco-2's described by source is recorded as Absence of HeLa cell contamination in 169 cell lines derived from human tumors^[7].
• Caco-2's described by source is recorded as Epithelial polarity, villin expression, and enterocytic differentiation of cultured human colon carcinoma cells: a survey of twenty cell lines^[8].
• Caco-2's described by source is recorded as Human tumor lines for cancer research^[9].
• Caco-2's described by source is recorded as Distinction of seventy-one cultured human tumor cell lines by polymorphic enzyme analysis.^[10].
• Caco-2's described by source is recorded as Presence of glycogen and growth-related variations in 58 cultured human tumor cell lines of various tissue origins.^[11].
• Caco-2's described by source is recorded as Beta-catenin mutations in cell lines established from human colorectal cancers^[12].
• Caco-2's described by source is recorded as Centrosome amplification and instability occurs exclusively in aneuploid, but not in diploid colorectal cancer cell lines, and correlates with numerical chromosomal aberrations^[13].
• Caco-2's described by source is recorded as APC mutations in sporadic colorectal tumors: A mutational "hotspot" and interdependence of the "two hits"^[14].
• Caco-2's described by source is recorded as Comprehensive galectin fingerprinting in a panel of 61 human tumor cell lines by RT-PCR and its implications for diagnostic and therapeutic procedures^[15].
• Caco-2's described by source is recorded as Spectral karyotype analysis of colon cancer cell lines of the tumor suppressor and mutator pathway^[16].
• Caco-2's described by source is recorded as Permeability characteristics of parental and clonal human intestinal Caco-2 cell lines differentiated in serum-supplemented and serum-free media^[17].
• Caco-2's described by source is recorded as The Caco-2 cell line as a model of the intestinal barrier: influence of cell and culture-related factors on Caco-2 cell functional characteristics.^[18].
• Caco-2's described by source is recorded as Analysis of P53 mutations and their expression in 56 colorectal cancer cell lines^[19].
• Caco-2's described by source is recorded as Identification by Real-time PCR of 13 mature microRNAs differentially expressed in colorectal cancer and non-tumoral tissues^[20].
• Caco-2's described by source is recorded as Cell growth, global phosphotyrosine elevation, and c-Met phosphorylation through Src family kinases in colorectal cancer cells^[21].
• Caco-2's described by source is recorded as Genomic and biological characterization of exon 4 KRAS mutations in human cancer^[22].
• Caco-2's described by source is recorded as 5-Fluorouracil response in a large panel of colorectal cancer cell lines is associated with mismatch repair deficiency^[23].
• Caco-2's described by source is recorded as Human embryonic stem cells and metastatic colorectal cancer cells shared the common endogenous human microRNA-26b^[24].
• Caco-2's described by source is recorded as Subtypes of primary colorectal tumors correlate with response to targeted treatment in colorectal cell lines^[25].
• Caco-2's described by source is recorded as Identification of a microRNA expression signature for chemoradiosensitivity of colorectal cancer cells, involving miRNAs-320a, -224, -132 and let7g.^[26].
• Caco-2's described by source is recorded as Epigenetic and genetic features of 24 colon cancer cell lines^[27].

Why It Matters

Caco-2 has Wikipedia articles in 5 language editions, a strong signal of global cultural recognition.^[2] Caco-2 is known by 5 alternative names across languages and contexts.^[28]