Unc-93 homolog B1, TLR signaling regulator
antigen processing and presentation of exogenous peptide antigen via MHC class II
The interaction between the ER membrane protein UNC93B and TLR3, 7, and 9 is crucial for TLR signaling
Proteolytic cleavage in an endolysosomal compartment is required for activation of Toll-like receptor 9
Mapping a dynamic innate immunity protein interaction network regulating type I interferon production
Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium
UNC93B1 delivers nucleotide-sensing toll-like receptors to endolysosomes
The Unc93b1 mutation 3d disrupts exogenous antigen presentation and signaling via Toll-like receptors 3, 7 and 9
Flt3 permits survival during infection by rendering dendritic cells competent to activate NK cells