EYA transcriptional coactivator and phosphatase 1
SIX1 mutations cause branchio-oto-renal syndrome by disruption of EYA1-SIX1-DNA complexes
Tyrosine dephosphorylation of H2AX modulates apoptosis and survival decisions
Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium
Biochemical and functional characterization of six SIX1 Branchio-oto-renal syndrome mutations
A human homologue of the Drosophila eyes absent gene underlies branchio-oto-renal (BOR) syndrome and identifies a novel gene family