# Christine Richardson

> academic, researcher, professor of biological science

**Wikidata**: [Q109981218](https://www.wikidata.org/wiki/Q109981218)  
**Source**: https://4ort.xyz/entity/christine-richardson


## References

1. [University of North Carolina at Charlotte](https://pages.charlotte.edu/christine-richardson/)
2. [International Standard Name Identifier](https://isni.org/isni/0000000137275831)
3. APE1 senses DNA single-strand breaks for repair and signaling
4. Bioflavonoids cause DNA double-strand breaks and chromosomal translocations through topoisomerase II-dependent and -independent mechanisms
5. Bioflavonoids promote stable translocations between MLL-AF9 breakpoint cluster regions independent of normal chromosomal context: Model system to screen environmental risks
6. Cell-free Xenopus egg extracts for studying DNA damage response pathways.
7. Oxidative stress, bone marrow failure, and genome instability in hematopoietic stem cells
8. Long-Term Effects of Chromatin Remodeling and DNA Damage in Stem Cells Induced by Environmental and Dietary Agents
9. Normal ABL1 is a tumor suppressor and therapeutic target in human and mouse leukemias expressing oncogenic ABL1 kinases
10. Rac2-MRC-cIII-generated ROS cause genomic instability in chronic myeloid leukemia stem cells and primitive progenitors
11. Aptamer-Functionalized Nanoparticles as “Smart Bombs”: The Unrealized Potential for Personalized Medicine and Targeted Cancer Treatment
12. Double-Strand Break Repair by Interchromosomal Recombination: An In Vivo Repair Mechanism Utilized by Multiple Somatic Tissues in Mammals
13. Dysregulation of AKT3 along with a small panel of mRNAs stratifies high-grade serous ovarian cancer from both normal epithelia and benign tumor tissues
14. Downregulation of HOXC6 in Serous Ovarian Cancer
15. Activating Notch1 mutations are an early event in T-cell malignancy of Ikaros point mutant Plastic/+ mice
16. Identification of Epithelial Ovarian Tumor-Specific Aptamers
17. MicroRNA miR-308 regulates dMyc through a negative feedback loop in Drosophila
18. Chromatin remodeling finds its place in the DNA double-strand break response
19. Etoposide-initiated MLL rearrangements detected at high frequency in human primitive hematopoietic stem cells with in vitro and in vivo long-term repopulating potential
20. Multipotent hematopoietic cells susceptible to alternative double-strand break repair pathways that promote genome rearrangements
21. Towards the uniform distribution of null P values on Affymetrix microarrays
22. Chromosomal translocation mechanisms at intronic alu elements in mammalian cells
23. RAD51, genomic stability, and tumorigenesis
24. Therapy-related acute myeloid leukemia-like MLL rearrangements are induced by etoposide in primary human CD34+ cells and remain stable after clonal expansion
25. The role of the DNA double-strand break response network in meiosis
26. Rad51 overexpression promotes alternative double-strand break repair pathways and genome instability
27. [LinkedIn](https://www.linkedin.com/in/christine-richardson-15365219/)
28. [University of North Carolina at Charlotte](https://sds.charlotte.edu/directory/christine-richardson/)