Biogenesis of lysosomal organelles complex-1, subunit 6, pallidin
positive regulation of natural killer cell activation
Hermansky-Pudlak syndrome type 7 (HPS-7) results from mutant dysbindin, a member of the biogenesis of lysosome-related organelles complex 1 (BLOC-1)
Cappuccino, a mouse model of Hermansky-Pudlak syndrome, encodes a novel protein that is part of the pallidin-muted complex (BLOC-1)
The dysbindin-containing complex (BLOC-1) in brain: developmental regulation, interaction with SNARE proteins and role in neurite outgrowth
The pallid gene encodes a novel, syntaxin 13-interacting protein involved in platelet storage pool deficiency
Phylogenetic-based propagation of functional annotations within the Gene Ontology consortium
BLOC-1 is required for cargo-specific sorting from vacuolar early endosomes toward lysosome-related organelles
Pigment mutations in the mouse which also affect lysosomal functions lead to suppressed natural killer cell activity
Platelet storage pool deficiency in mouse pigment mutations associated with seven distinct genetic loci
The schizophrenia susceptibility factor dysbindin and its associated complex sort cargoes from cell bodies to the synapse
Interaction of the murine dilute suppressor gene (dsu) with fourteen coat color mutations